Australia cemetery
to offer carbon-free funerals
CANBERRA,
Mar 11: An Australian cemetery has unveiled
plans to take the carbon out of cremations by
offering new green funerals to help combat global
warming.
On the day
Australia's formal ratification of the Kyoto
Protocol on Greenhouse emissions comes into
force, the Centennial Park cemetery in the South
Australian state capital of Adelaide said it had
studied the carbon impact of burials and
cremations.
While cremations
initially produce more carbon emissions than a
burial, cemetery chief executive Bryan Elliott
said over time, burials ended up producing about
10 per cent more greenhouse gas.
''If we plant one
tree for every service, either burial or
cremation, we will more than offset the carbon
emissions,'' Elliott told Reuters today.
The Centennial
Park Cemetery carries out more than 900 burials
and around 3,300 cremations a year. Elliott said
every cremation created around 160 kg of carbon
dioxide, compared to 39 kg of carbon dioxide for
each burial.
But when the cost
of maintaining grave sites, mostly covered by
lawns at Centennial Park, is taken into account,
cremations came out 10 per cent greener than
burials.
''This is because
we must look after the gravesite for a number of
years by watering and mowing the surrounding lawn
area and maintaining the concrete beam on which
the headstone is placed,'' Elliott said.
''Burial is a more
labour and resource intensive process, consumes
more fuels and produces larger quantities of
waste than cremation.''
The move was
prompted by local calls for natural burials,
where a tree is planted over a grave. But those
proposals would not work in a major suburban
cemetery, where space is limited and graves can
be re-used, Elliott said.
In South
Australia, graves are leased for only 50 years,
and they can then be re-used if relatives do not
the renew the lease.
Elliott said the
cemetery planned to bear the cost of carbon
offsets and would not charge more for funerals as
it attempts to cut its greenhouse emissions,
blamed for global warming. (AGENCIES)
Japan seeks new
form of flu vaccine, investors jump
TOKYO,
Mar 11: A group of Japanese researchers has
developed a substance that could potentially help
make flu vaccines effective for multiple strains
of the disease, including strains of the bird flu
virus, Japan's National Institute of Infectious
Diseases said.
The substance
faces a lot more testing but investors seized on
media reports of it yesterday, pushing the shares
of a chemical firm involved in the project, NOF
Corp, up nearly 21 percent.
Traditional flu
vaccines create antibodies which act against flu
viruses, but since virus surfaces frequently
mutate, different vaccines have to be made every
year.
The group found
that when a peptide derived from the influenza
virus is induced into mice, it could act against
cells infected by multiple strains of influenza,
including bird flu.
Part of the
research was reported in the Journal of
Immunology in 2006, and the group presented its
findings last month at Japan's National Cancer
Center. The only tests so far have been on mice.
The next step is
to develop a vaccine that works against multiple
strains of flu and is proved safe for humans,
said Tetsuya Uchida, a senior investigator at the
National Institute of Infectious Diseases.
''It usually takes
about five years to develop vaccines for clinical
use. But bird flu is an emerging issue and we
would like to develop this as soon as possible,''
Uchida said.
The findings could
also potentially be applied to create drugs to
treat AIDS, tumours and other diseases, he said.
(AGENCIES)
Where ever you
click... The network follows
SYDNEY,
Mar 11: Internet has made world ''a global
village'' and now every nook and corner of that
village is being spied by large web companies to
earn big bucks.
According to new
statistics of online consumer data, web companies
are learning more than ever about what users
search on the internet, gathering clues about
their tastes and preferences several hundred
times a month.
The companies use
the information to predict what content and
advertisers people want to see. They can charge
steep prices for carefully tailored ads because
of their high response rates.
Earlier also,
these practices of internet companies were
highlighted but only vague statistics were
available.
The new analysis
indicates that web companies are take the trails
people leave behind as they move around the
internet, analysing them and anticipate their
next steps. So anybody who searches for
information on topics as iron supplements,
airlines, hotels and soft drinks may see ads for
those products and services later on.
''When you start
to get into the details, it's scarier than you
might suspect,'' Executive director of the
Electronic Privacy Information Centre, Mark
Rotenberg, said, adding, ''We're recording
preferences, hopes, worries and fears.''
Web companies,
however, claim that have policies in place to
protect consumers' names and other personal
information from advertisers. Moreover, the data
is a boon to consumers, as it makes the ads they
see more relevant.
People who spend
more time on the internet will have more
information transmitted about them. The comScore
per person figures are averages; occasional web
users have far less transmitted about them.
(UNI)
Snakes win in
creepy-crawly arms race: Study
WASHINGTON,
Mar 11: Garter snakes have won out in a
toxic arms race with their favored prey -- newts
--researchers said in study that challenges
conventional thinking on evolution.
They said their
report helps demonstrate how evolution can move
very quickly, and said it could also shed light
on human disease, itself a kind of toxic arms
race.
''These kinds of
arms races are one of the models that we use for
understanding host-parasite evolution and even
viral disease evolution,'' Charles Hanifin of
Utah State University, who led the study, said in
a telephone interview yesterday.
His team studied
rough-skinned newts, a type of salamander, found
on the west coast of the United States and
Canada. They are among the most poisonous known
animals, carrying a lethal load of paralyzing
tetrodotoxin, known as TTX for short.
''Because they
have this toxin, nothing else eats them,''
Hanifin said.
Just a little bit
of TTX can kill thousands of mice, but certain
garter snakes are immune and gobble the newts
with no apparent ill consequences. But not all of
them.
''Both toxicity of
newts and resistance of snakes vary
geographically. Where newts are absent or
nontoxic, T. Sirtalis (garter snakes) are not
resistant to TTX,'' Hanifin's team wrote in their
report, published in the Public Library of
Science journal PLoS Biology.
YOUR DINNER OR
YOUR LIFE
The usual thinking
in evolution is driven by the so-called
''Life-Dinner'' principle. The idea is that prey
animals get a little faster, or in this case a
little more poisonous, to stay one step ahead of
their predators.
''In these kinds
of situations you would expect the prey to evolve
more quickly and strongly than the predator,''
Hanifin said. This is because the prey stands to
lose more.
''If the prey
loses that interaction, the prey is dead. But if
the lion doesn't catch the antelope, then the
lion is not dead but has suffered the loss of a
dinner,'' Hanifin said.
''That obviously
is the complete opposite of what we see here.''
Hanifin's team
found in 10 different places that garter snakes
had evolved immunity to TTX, but the newts never
got poisonous enough to escape the snakes.
This immunity to
TTX boiled down to a small change in a single
gene in the snakes, Hanifin said.
In places where
the newts were not poisonous, the snakes did not
have the genetic change -- showing that it did
evolve in response to the poisonous varieties of
newt. There were no places where the newts were
toxic enough to escape the snakes.
The discovery
shows evolution can make sudden leaps. ''Boom --
all of a sudden there is no arms race any more,''
Hanifin said.
It could explain
other cases of extreme evolution -- such as
hummingbirds that have grown long beaks to reach
deep into flowers for nectar, Hanifin said.
And it would
explain quick cases of evolution -- such as the
emergence of antibiotic-resistant bacteria among
people. (AGENCIES)
If both parents
have Alzheimer's, your risk soars
WASHINGTON,
Mar 11: If both your parents have
Alzheimer's disease, you probably are more much
likely than other people to get it, researchers
said yesterday.
Their study
focused on 111 families in which both parents
were diagnosed with Alzheimer's disease, the most
common form of dementia among the elderly, and
assessed the risk for developing it among the
offspring.
The parents had
297 children who lived into adulthood. Of the 98
men and women who were at least 70 years old, 41
of them -- about 42 percent -- developed
Alzheimer's disease, researchers at the
University of Washington in Seattle found.
''That's greater
than you would expect in the general population
in that age group,'' Dr Thomas Bird, one of the
researchers, said in a telephone interview
yesterday.
In the general
population, risk for the disease begins to rise
at about age 65, with the number of people
developing the disease doubling every five years
beyond that, experts say.
But about
two-thirds of the adult offspring in the study
still had not reached age 70. Counting all 297 of
these adult offspring regardless of age, 23 per
cent already had been diagnosed with Alzheimer's
disease, with the disease diagnosed on average at
age 66, the researchers found.
Bird said that
compares to the roughly one in 10 chance that the
average person will develop the disease.
''I think it
confirms that there's a strong genetic component
in the disease and that's not a surprise,'' said
Bird, whose study was published in the Archives
of Neurology.
Scientists do not
yet fully understand the causes of Alzheimer's
disease, although genetics plays an important
role. There is no cure.
Bird said there is
only one gene, known as ApoE, that is generally
agreed among researchers as a risk factor for the
disease but there likely are many more.
The ApoE gene is
involved in making a substance in the body that
helps carry cholesterol in the bloodstream and
the gene seems to influence the age of onset of
Alzheimer's.
The researchers
have been doing the study for about two decades
and intend to continue for at least another
decade.
''The numbers will
be interesting to follow as they get older and
older,'' Bird said.
Bird said the
study is not examining the Alzheimer's risk for
people who have one but not two parents who
develop the disease.
In order to
confirm that both parents actually had
Alzheimer's, the researchers reviewed the medical
records and in many cases the brain autopsies of
those who had died, and tried to meet in person
to assess those who still living.
In people with
Alzheimer's disease, healthy brain tissue
degenerates, causing an inexorable decline in
memory and mental abilities. The average length
of time from diagnosis to death is about eight
years.
(AGENCIES)
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